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Additionally, Tudor domain-containing JMJD2A can antagonize 53BP1 recruitment to DSB sites by binding to demethylated H4K20, but RNF8 and RNF168 trigger JMJD2A ubiquitination and degradation to facilitate 53BP1 foci formation (Mallette et al

(2011) and Yilmaz-Ersan et al

The practical implication is uncomfortable but honest: a 12 week window is a handling convention, not a measured stability specification for any particular vial, and degradation is not something that can be seen in a clear solution