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Moreover, the in vivo results demonstrated the ability of GliSODin to reduce differentiated Th2 and Th17 cell reactivation by reducing their proliferation, confirming previous studies showing that ROS upregulates costimulatory surface molecules CD40, CD86, and MHCII and promote T-cell proliferation (32)

It is a prodrug, which requires the Mtb catalase-peroxidase KatG activation to generate its acyl radical or acyl anion form [67], which subsequently reacts with the cellular NAD + , resulting in an INH-NAD adduct and consequently interferes with the biosynthesis of mycolic acids

Thus, the inhibitors of histone deacetylation or demethylation may be promising agents for MS treatment

doi: 10.3390/biom12091216 18