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For instance, during the early phases of acute myocardial infarction, the acute onset of high-grade local inflammation is revealed not only by the polymorphonuclear neutrophils activation and recruitment of mononuclear cells but also by the local release of pro-inflammatory molecules such as IL-1 and IL-6 [118]

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This may subsequently induce alterations in local gut microbial structure and metabolic products, thereby establishing and exacerbating a vicious cycle of ferroptosis