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Group I was induced with TBI and treated with DMSO at 67.5 mg/kg orally once daily which started 30 min after the induction of TBI and lasted 21 days

( h ) The ubiquitination of nuclear IDO1, the binding of IDO1 and KEAP1 in synchronized MCF-7 cells at different time points was analyzed by Western blot

From myocardial I/R injury to DCM, atherosclerosis, and HF, cuproptosis mediates pathogenic processes through mechanisms including mitochondrial dysfunction, proteotoxic stress, GSH depletion, and lipid metabolism disruption