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Description

Oxidative stress is involved in the pathophysiology of PD as excessive production of reactive oxygen species (ROS) contributes to dopaminergic neuronal damage, which is susceptible to oxidative injury due to high metabolic activity and dopamine metabolism

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After treatment with inhaled doses of NAC (1 ml of 200 mg) arterial blood oxygen saturation, HCO level, and shunt fraction were significantly improved compared to those of the SM exposure