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This speculation is also supported by data from previous studies which showed that EXOs are potential messenger units for ageing skeletal muscle regeneration and the improvement of Huntingtons disease in symbiotic blood exchange systems [22, 46]

Patra JK, Das G, Fraceto LF, Campos EVR, Rodriguez-Torres MDP, Acosta-Torres LS, et al

Interestingly, non-oxidative dopamine was reported to potently inhibit erastin-induced ferroptosis in both cancerous and non-cancerous cells by stabilizing GPX4, 218 suggesting that dopamine may be a promising candidate drug for alleviating ferroptosis-related tissue injury and disease, as well as certain neurodegenerative diseases