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Abstract As a chronic autoimmune disease with complex aetiology, rheumatoid arthritis (RA) has been demonstrated to be associated with mitochondrial dysfunction since mitochondrial dysfunction can affect the survival, activation, and differentiation of immune and non-immune cells involved in the pathogenesis of RA

This review presents the latest advances of insulin-loaded nanoparticles, including PLGA, HA, gelatin, chitosan, alginate, and -PGA/-TCP

However, future studies should explore whether inhibiting these pathways could introduce off-target effects or interfere with other essential cellular processes