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SLC9A3R1 and NAT9 are regulators of signal transduction, the immunologic synapse, and T-cell proliferation (Krueger and Bowcock 2005)

These pieces of evidence suggest that the eCB system is a vital component in the response to stress

Cathepsin G in NETs activates complement C5, generating C5a, which enhances neutrophil NADPH oxidase activity through the C5aR1 receptor, establishing a prothrombotic-proinflammatory feedback loop (112, 113)