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ber drei pharmakologische Hauptmechanismen VEGF-Aktivierung mit Angiogenese-Frderung (Neubildung von Blutgefen in geschdigtem Gewebe), Stickstoffmonoxid-Signalweg-Aktivierung fr Mikrozirkulation und anti-entzndliche Wirkung, sowie Wachstumsfaktor-Modulation (EGR-1, FAK) fr Zell-Migration und Geweberegeneration

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Copper transport protein antioxidant-1 promotes inflammatory neovascularization via chaperone and transcription factor function