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These diverse molecular mechanisms collectively demonstrate that EED-induced reproductive toxicity results from systematic disruption of multiple, interconnected biological pathways rather than simple receptor binding events

doi: 10.3390/ijms22010207 37 AlaeeSIlaniM

2021;320:C216-216C224

Mechanistically, the activated MLKL can be localized in mitochondria, which may promote the formation of mitochondrial pores and the efflux of mtDNA into the cytoplasm, thereby promoting the activation of the cGAS-STING pathway in tumor cells after RT [282]