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Chronic marginal iron intakes during early development in mice alter brain iron concentrations and behavior despite postnatal iron supplementation

Cells were then subjected to ROS level measurement using DCFH-DA probe, inflammatory cytokine detection using ELISA, and M1 polarization analysis using flow cytometry, as described above

Cerebrolysin reduces bloodcerebrospinal fluid barrier permeability change, brain pathology, and functional deficits following traumatic brain injury in the rat

These deficiencies lead to the accumulation of unrepaired DNA lesions, driving cell dysfunction and genomic instability, which further promotes ARC progression (108)