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Eur J Biochem 159 : 415423 Snyder R, Chepiga T, Yang CS, Thomas H, Platt K, Oesch F 1993 Benzene metabolism by reconstituted cytochromes p450 2B1 and 2e1 and its modulation by cytochrome b5, microsomal epoxide hydrolase, and glutathione transferases: evidence for an important role of microsomal epoxide hydrolase in the formation of hydroquinone

The analysis suggests that the risk of APAP-induced hepatotoxicity is increased if APAP is ingested shortly after alcohol is cleared from the body in chronic alcohol users

Depletion of GSH as a consequence of acetaminophen toxicity to the liver has attracted the most attention in the medical scientific community, as it can frequently be fatal or require a liver transplant or emergency treatment to prevent liver failure (the liver is the organ with the greatest concentration of GSH)

The significance of the gap junction in the bystander effect has been questioned by several investigators in recent years