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These findings suggest that TLR/PI3K/AKT-activated EPRS1 phosphorylation at Ser999 directs the first line of anti-inflammatory immune defense against inflammatory signals, including bacterial infection, while IFN-mediated EPRS1 phosphorylation at Ser999 shapes translational fine tuning to protect cells from inflammation and injury in the presence of persistent inflammatory stimuli

Studies have shown that TSA downregulates extracellular matrix (ECM) gene transcription and collagen expression in dermal fibroblasts by inhibiting the TGF-/Smad and MAPK/ERK signaling pathways, contributing to its antifibrotic effects (60,61)

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