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We hypothesized that low glutathione redox status would be associated with elevated markers of oxidative protein and DNA damage, inflammation and mitochondrial superoxide production in two regions that have been reported to be abnormal in autism, cerebellum and BA22

Int J Mol Sci 22(19):10493 Piacenza L, Trujillo M, Radi R (2019) Reactive species and pathogen antioxidant networks during phagocytosis

S-Acetyl-L-glutathione (50 M) increases intracellular GSH levels in primary fibroblasts derived from patients with glutathione synthetase deficiency.1 It induces apoptosis in Daudi, Raji, and Jurkat lymphoma cells when used at a concentration of 5 mM

To begin to evaluate potential targeting of c-MYC in endometriosis therapy, we utilized the well-characterized human endometriotic epithelial cell line, 12Z which expressed eGFP (Figure 2 depicts representative immunofluorescence for Omomyc localization with white arrows highlighting nuclear expression of Dox-induced Omomyc expression