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Description

Such disruptions can impair mitochondrial protein import and activate the UPRmt, a conserved pathway that restores mitochondrial homeostasis (Melber and Haynes, 2018

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Taken together, these experiments point to extracellular determinants for melanogenesis and support the conclusion that extracellular changes in gsh2 mutants lead to a redox environment incompatible with melanin formation

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Bioprinted osteogenic and vasculogenic patterns for engineering 3D bone tissue

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FESEM and TEM confirm the influence of chitosan on BiNP dimensions and morphology The FESEM was employed to elucidate the morphological characteristics and average dimensions of Bi@Chi-DEC NPs

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