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On the other hand, endothelial STIM1 plays a critical role in the activation of eNOS to produce NO, such that EC-specific deletion of the STIM1 gene impairs endothelium-dependent vasorelaxation and increases blood pressure (Nishimoto et al., 2018)

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doi: 10.3389/fmolb.2021.660959 Received 30 January 2021 Accepted 08 April 2021 Published 17 May 2021 Volume 8 - 2021 Edited by Venkaiah Betapudi, Chemical and Biological Defense Division, United States Department of Homeland Security, United States Reviewed by Alexey Moskalev, Institute of Biology, Komi Scientific Center (RAS), Russia