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This is in line with previous research suggesting that differences in CPD for AA and non-AA smokers may be explained through biological markers as AA smokers have a slower rate of nicotine metabolism [37] and that smokers who have a faster rate of nicotine metabolism smoke more cigarettes per day [38]

Although direct causality remains contested within evidence-based tobacco policy discussions, these country-level experiences suggest that product differentiation , when combined with robust youth protection strategies and tobacco-control measures , may contribute to more significant and sustained reductions in smoking prevalence

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Countries like the United States and Canada implement regulations that promote harm reduction, enhancing market growth