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Unlike the molecular design of brensocatib and BI 1291583, HSK31858 employs a non-peptide non-covalent inhibition mechanism, binding to DPP1 through non-covalent interactions with an IC50 of 57.4 nM ( Structural biology research reveals the unique binding mode of HSK31858 with DPP1

Yokota T, Nishio H, Kubota Y, Mizoguchi M

however, we have previously reported beneficial effects of ELAM treatment on mitochondrial function and heart and muscle physiological function in female mice [12,13,14]