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It seems likely that the spike in IL-6 , secreted by the macrophages responding to the viral entry, triggers NETosis in the patients with severe COVID-19, leading to a complex interaction among NETosis, complement and coagulation pathways [18], pulmonary immune thrombosis and acute respiratory distress

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These insights have not only enhanced our understanding of CHD pathophysiology but have also established a theoretical foundation for developing ferroptosis-targeted therapeutic strategies

In a double-blind, placebo-controlled randomized trial, Hendouei and colleagues evaluated resveratrol (non-botanical resveratrol