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Spinocerebellar ataxias (SCAs) represent a genetically heterogeneous subset of autosomal dominant progressive neurological disorders predominantly caused by expansion of a glutamine-encoding CAG trinucleotide repeat, leading to the accumulation of misfolded proteins, dysfunction of ion channels and mitochondria, and ultimately neurodegeneration, with PCs, brainstem, and spinal cord neurons being particularly affected in SCA type 1 (Watase et al., 2002

(34457354) The use of steroids in fat embolism syndrome is controversial, with some evidence suggesting that steroids reduce the incidence of hypoxemia

Genes Dev 12:557570 Moreira S, Stramer B, Evans I, Wood W, Martin P (2010) Prioritization of competing damage and developmental signals by migrating macrophages in the Drosophila embryo