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In cells, oxidative stress generated by Reactivating p53 and Inducing Tumor Apoptosis (RITA) induces TrxR1 tetramerization and covalent linkage, resulting in reduced activity or inactivation of TrxR1 (Xu et al., 2015)

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They generate the vast majority of our cellular energy in the form of adenosine triphosphate (ATP)

Inflammatory cytokines and enzymes, such as interleukin-6 (IL-6), interleukin-17 (IL-17), tumor necrosis factor-alpha (TNF-), nitric oxide (NO), inducible nitric oxide synthase (iNOS), and matrix metalloproteinases (MMPs), show a high correlation with the pathogenesis of hemorrhoids