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In contrast, fetal muscle showed no changes in mitochondrial activity but displayed reduced expression of genes involved in fat metabolism and energy production, suggesting early transcriptional changes that could affect postnatal muscle energy utilization (Safain et al., 2025)

[1] [2] It functions primarily within the mitochondriathe cellular powerhouseswhere it participates in the electron transport chain to generate adenosine triphosphate (ATP), the body's primary energy currency

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Vigorous shaking creates mechanical stress that promotes aggregation, the clumping of peptide molecules that reduces potency