does oxidation state of glutathione depends on cysteine residue Structural and functional fine mapping cysteines in mammalian glutaredoxin reveal their differential susceptibility Oxidative post-translational modification of cysteine

Cyss uptake by SLC7A11/xCT induces NRF2 expression and activation Since TNBC cells are known to express high levels of the cystine/glutamate antiporter SLC7A11/xCT, we wondered whether the cellular Cyss uptake and consequently cytoplasmic Cys levels are responsible for the activation of the NRF2 pathway

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Notably, the HG-3 cells had a relatively high basal level of autophagy

One advantage that the generation of pluripotent stem cells from somatic cells brings is the possibility to cultivate patient-derived cells and to study the consequences of the disease they may carry in different cell types in a patient-specific manner