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A study on genetic linkages between aberrant lipid metabolism and PD directly demonstrates that glucocerebrosidase and sphingomyelin phosphodiesterase 1 mutations enhance PD risk in humans, and that galactosylceramide beta hydrolase function loss increases -synuclein aggregation and accumulation in experimental murine models

The Government will seriously follow up on the compliance of compulsory testing notices by persons subject to compulsory testing, and continually review the compulsory testing arrangement of the above specified areas taking into account the epidemic development

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