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This review describes the potential role of SGLT2 inhibitors as a breakthrough therapeutic option in PD, emphasizing their ability to address unmet clinical needs and improve patient outcomes

Researchers have attempted to elucidate the underlying mechanisms of DCM, which are usually treated as determinants of uncontrollable persistent pathological changes, such as aberrant hyperglycemia, insulin resistance, excessive oxidative stress, inflammatory response, and mitochondrial dysfunction [7,8,9]

Each compound addresses distinct physiological challenges associated with metabolic optimization, and together they create a more comprehensive approach than either achieves independently

SLC7A11 inhibitors represent a promising therapeutic target by facilitating the induction of ferroptosis in breast cancer