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This suggestion was further insisted as a potential mechanism of TB-500 by the authors of the research study, who also commented that "transfection of anti-miR-146a nucleotides reversed the inhibitory effect of T4 on IRAK1 and TRAF6." Thus, TB-500 also appears to potentially work for healing by exerting anti-inflammatory action.(6)

A subsequent SEC-based thermal shift assay using purified proteins quantified this stabilization, showing the tagged hTauT had a melting temperature ( T m ) of 82 C, significantly higher than the 46 C observed for untagged hTauT (Supplementary Fig

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