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A specific oxidoreductase, previously known as apoptosis-inducing-factor mitochondrial-2 (AIFM2), capable of recycling reduced ubiquinol (Co-enzymeQ 10 H 2 ) from ubiquinone at the expense of NAD(P)H, has been presented as a potential ferroptosis inhibitor due to the fact that its overexpression complements the loss of GPx4 in PFA1 and human fibrosarcoma (66, 67)

A total of 70 male SpragueDawley rats were systematically allocated into seven distinct groups, inclusive of control and three dosage categories of Rhodiola extract (1.5, 3, and 6 g/kg)

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