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When compared to experimental and clinical data on the accumulation of the byproducts of acetaminophen metabolism, APAP-S, APAP-G, and NAPQI-GSH, in the plasma and in the urine of humans, the model gives accurate predictions (see Figures 2, 3, 4 and 5)

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Metabolic syndrome and insulin resistance as risk factors for development of chronic kidney disease and rapid decline in renal function in elderly

By mapping these interactions, we were able to visualize how alterations in key metabolic pathways are linked to each other and to potential molecular targets, providing insight into the coordinated metabolic response induced by the treatment