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Rapamycin, a macrolide immunosuppressant generated by the bacterium Streptomyces hygroscopicus, impedes the transition of T cells from the G1 phase to the S phase of the cell cycle by inhibiting IL-2-mediated signaling pathways, thus exerting anti-inflammatory activity [260, 261]

The outcomes demonstrated that MCF7 cells (5 g/mL) were specifically cytotoxic to the dichloromethane extract, but MCF 10A (non-cancerous breast) cells were not considerably inhibited