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Description

Effect of high-dose N-acetylcysteine on exacerbations and lung function in patients with mild-to-moderate COPD: a double-blind, parallel group, multicentre randomised clinical trial

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The mechanisms typically involve chromosomal translocations, aneuploidy, or immunosuppression-induced lymphoproliferation [140, 142]

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Diffuse plaques associated with astroglial amyloid protein, possibly showing a disappearing stage of senile plaques

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