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Claudin-1-dependent destabilization of the bloodbrain barrier in chronic stroke

However, it is essential to ensure that the reprogramming process and subsequent culture conditions do not introduce genetic abnormalities or mutations that could compromise the safety and efficacy of iPSC-based therapies [1, 7]

On day 4, the protein expression of key DE markers, SOX17 and forkhead box protein A2 (FOXA2), were evaluated by immunocytochemistry staining on 4% paraformaldehyde (PFA)-fixed cells using SOX17 mouse anti-human monoclonal antibody (Abcam) and FOXA2 rabbit anti-human monoclonal antibody (Abcam), as described below