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Mov Disord 33(5):730741 Kazlauskaite A, Kondapalli C, Gourlay R, Campbell DG, Ritorto MS, Hofmann K, Alessi DR, Knebel A, Trost M, Muqit MM (2014) Parkin is activated by PINK1-dependent phosphorylation of ubiquitin at Ser65

CLBP arises from a variety of etiological mechanisms, which can be broadly grouped according to the presence or absence of identifiable structural pathology [1]

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For example, the facultative anaerobe Enterococcus faecalis harbors a coenzyme A persulfide reductase (CoAPR) that mitigates the toxic effects of an accumulating coenzyme A persulfide (CoASSH) during sulfide stress by reducing the persulfide to coenzyme A and H 2 S, which is likely further metabolized 16