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ApoE represents a significant risk factor for VD, with meta-analyses demonstrating heightened susceptibility to VD in individuals with the ApoE4 allele compared to those with the ApoE3 allele (Yin et al., 2012)

They cause damage to vessels through by increasing the number of thrombins, causing NOX activation and elevated production of ROS, which leads to DNA and lipid oxidation, concurring to atherosclerosis [57], which has also been confirmed in Jagadeesha et al.s [44] research

The proposed fat loss mechanism operates through SAM and NAD+ preservation

Burst frequencies were measured at baseline and again following sequential Glu (expected to increase excitatory neuron activity) and AP5 (expected to reduce excitatory neuron activity) treatments in PBS, Con and AD-tau cultures using the recordings described above (Fig