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STW3-VI pre-treatment lead to a concentration-dependent abolishment (of glutamate/NMDA-induced decrease of viability) by 25% ( p 0.01) and 50% ( p 0.01) at 10 g/ml STW3-VI in comparison to 0.1 mM glutamate (Figure 3A) or 0.1 mM NMDA (Figure 3B) treatment, indicating a cytoprotective effect of STW3-VI against glutamate- or NMDA-induced neurotoxicity

Intervening and treating oxidative stress is one of the most effective techniques for improving the pathogenetic status of ASD patients

Necrotic apoptosis is a form of programmed necrosis regulated by genetic programming, and its core executive mechanism relies on the activation of the receptor-interacting protein kinase 1 (RIPK1) -RIPK3-MLKL signaling axis (Degterev et al., 2005

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