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However, impairment begins at much lower levels

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Release of proangiogenic factors and increased hypoxia in response to vascularization blockade can lead to recruitment of endothelial progenitor cells (EPC) from the bone marrow, which contribute to tumor vascularization and have been linked to development of resistance to anti-VEGF therapy [303]

The pairing is biologically reasoned and widely used in research settings, but it has not been tested as a combination in a controlled human study