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Introduction -thalassemia is the most common inherited disease, characterized by decreased or absent -globin chain synthesis and hemoglobin A production ( The incidence of myocardial iron overload in transfusion-dependent -thalassemia patients has increased from 11.4%15.1% in early studies to 26.1%36.7% in recent studies ( As iron loading progresses, the capacity of transferrin to bind and detoxify iron is eventually exceeded, leading to non-transferrin-bound iron in plasma, which promotes oxidative stress, mitochondrial dysfunction, and ferroptosis ( Mechanisms of myocardial injury caused by iron overload Studies have shown that iron myocardial overload causes myocardial dysfunction

5g), but the growth rate was slightly different (optical density of controls at 72 h GLC/GAL = 1.6), given that galactose culture makes cells divide more slowly 47 , meaning that further controls are needed to confirm the role of mitochondrial metabolism in modulating sensitivity

Derived from a protein found in human gastric juice, purpose-built for the gastrointestinal lining

In an attempt to observe this transcriptional interaction, a cell free experiment was attempted