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Use of APAP in babies and small children was not monitored as medical practice changed To establish any association between the use of APAP in the pediatric population and the incidence of neurodevelopmental disorders, it is most convenient to establish the prevalence of both factors over time with some degree of certainty

TNFR1-JNK signaling is the shared pathway of neuroinflammation and neurovascular damage after LPS-sensitized hypoxic-ischemic injury in the immature brain

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We have shown that Set7/9 attenuates MDM2 expression and suppresses MDM2 functions under DNA damage conditions 151 (Figure 4B)