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4.1.2 Prolyl hydroxylase activator In the hypoxic microenvironment, PHD activity is inhibited, resulting in high expression of HIF- and participating in pathological processes such as RA inflammatory response, angiogenesis, and bone destruction by affecting downstream target genes

In this case also the increase in TSH level was not statistically significant between the groups

Genetic or pharmacological blockade of Parkin abolishes its benefit, underscoring mitophagy as the critical driver ( Urolithin A, a gut-derived ellagitannin metabolite, similarly boosts PINK1-Parkin-dependent mitophagy, thereby dampening ROS-mediated NLRP3 priming, Caspase-1 cleavage and GSDMD-driven pyroptosis