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Acrolein as an electrophile can induce the activation of Nrf2 independently of NADPH oxidase, by depletion of GSH [32], by limiting the enzymatic activity of thioredoxin, peroxiredoxins and other Nrf2 transcription targets, through adduct formation [70] and by potentially reacting with KEAP1 cysteine residues, promoting Nrf2-KEAP1 dissociation [31] [4]

Enhanced assembly of these supramolecular structures demonstrates significant optimization of mitochondrial bioenergetics and augmentation of exercise performance (Champagne et al

doi: 10.3389/fncel.2022.889765 Received 04 March 2022 Accepted 19 April 2022 Published 19 May 2022 Volume 16 - 2022 Edited by Thorsten Roland Doeppner, University Medical Center Gttingen, Germany Reviewed by Fabien Gosselet, Universit dArtois, France