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Microbiota, gut hormones, and metabolic processes EECs are distributed along the length of the intestinal epithelium and serve as the major cell type for nutrient sensing and trigger subsequent signaling through the release of gut hormones (Fig

F-53B, a chlorinated alternative to perfluorooctane sulfonate (chemical name: 6:2 chlorinated polyfluorinated ether sulfonate), has been used as a replacement compound

Copper deficiency may thus impair the function of these enzymes, leading to an impaired antioxidant defense system and increased susceptibility to oxidative stress [70, 71]

Several new therapeutic approaches aimed at slowing the progression of PD are considered disease-modifying treatments and include anti-inflammatory agents (selnoflast, azatriopine), antioxidants, agents that stimulate mitochondrial function, agents that increase glucocerebrosidase activity (ambroxol), agents that activate the glucagon-like peptide-1 receptor (exenatide, lixisenatide, Peptron), agents that block specific kinase activity (SPARCs K0706, Inhibikases IKT148009, and Il-Yangs radotinib), agents that reduce or inhibit LRRK2 activity (BIIB122), drugs that target GI tract activity (rifaximin), and molecules that prevent -syn aggregation or disaggregation of existing complexes (prasinezumab, memantine, buntanetap)