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For example, although one report suggests that NOS inhibition does delay fatigue in electrically stimulated diaphragm muscle (128), other studies conclude that NO does not directly influence skeletal muscle fatigue resulting from prolonged submaximal contractions (74, 221)

Moreover, dihydroartemisinin may decrease the expression levels of the key G0/G1 regulators, CDK2/4, and cyclin E1 through the mTOR/HIF-1 signaling, thereby inducing A549 cell cycle blockade and alleviating gefitinib resistance (Hu et al., 2023