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In conclusion, our study found a high prevalence of hypovitaminosis D in COVID-19 patients, suggesting a possible role of low VD status in increasing the risk of COVID-19 infection and subsequent hospitalization

When iron load increases (e.g., elevated iron stores, high serum iron levels) [26], elevated hepcidin directly binds to membrane-bound FPN, promoting its internalization and degradation within intestinal epithelial cells, thereby inhibiting iron absorption into the bloodstream [27]