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demonstrated that QE reversed the inhibition of transcription factor EB (TFEB) nuclear translocation incited by ethanol and exhibited similar effect to Torin 1 mTOR activity inhibitor) which could promote TFEB nuclear translocation, indicating that QE could alleviate chronic ethanol consumption induced liver injury and autophagic flux suppression by regulating mTOR-TFEB pathway (Li et al., 2019)

Figure 1 Table 1 Expression changes and functional outcomes of key ferroptosis-related molecules in COPD

This vulnerability stems partly from their antioxidant defensesparticularly in the liver and red blood cellsbeing somewhat more limited than those of other animals

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