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Increased g-ECS activity may result from transcriptional or post- transcriptional changes (May et al

In animal studies, injected BPC-157 has shown potential effects in models of tendon rupture, ligament injury, muscle damage, nerve injury, and spinal cord trauma

By curbing excessive glutamate activity, DSIP may protect neurons from excitotoxicity, a mechanism relevant to its benefits in alcohol withdrawal (where glutamate rebound is a primary cause of seizures)