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Elevated expression of AARS1 and lactylation of p53 are often associated with poor prognosis of patients, and administration of -alanine to antagonize the lactylation of p53, coupled with doxorubicin to activate the transcriptional activity of p53, can significantly inhibit the proliferation of tumor cells in animal models [23]

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Our current findings revealed that blockade of COX-1 enzyme as well as treatment with the non-specific COX-inhibitor indomethacin reversed the effects of NPW on neutrophil recruitment and antioxidant capacity, suggesting that COX-1 enzyme activity mediates the therapeutic effect of NPW on gastric oxidative damage

doi: 10.1016/j.clinmicnews.2008.01.006