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Dihexa was engineered for oral bioavailability through modifications that resist proteolytic degradation and facilitate blood-brain barrier penetration via passive diffusion

The MetHGF interaction regulates various signalling pathways involving downstream kinases, such as Akt and Erk

Ultimately, we advocate a rationale to improve the outcome of anti-MET therapies on the basis of thorough consideration of the entire spectrum of MET-mediated biological responses, which implicates adequate patient stratification, meaningful biomarkers and appropriate clinical end points