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Obstruction of GSH synthesis affects the lipid membrane repair ability of GPX4, increases the rate of toxic lipid free radical and ROS accumulation, and promotes lipid peroxidation, resulting in ferroptosis [1, 2]

Interestingly, when comparing the modulation of tg expression in both PTU-treated WT and nrf2a5 homozygous embryos, we observed a marked reduction in tg expression in nrf2a5 homozygous mutant embryos (Fig 2V)