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2.2.4 FSP1 and other emerging regulatory factors Alongside the critical role played by the GPX4 system, the ferroptosis suppressor protein 1 (FSP1)-coenzyme Q10 (CoQ10) pathway independently regulates ferroptosis ( Additionally, recent findings have identified GTP cyclohydrolase-1 (GCH1), dihydroorotate dehydrogenase (DHODH), and several other factors as ferroptosis regulators, further expanding the ferroptosis network and offering new directions for elucidating ferroptosis in OA ( 2.3 Overview of ferroptosis-associated changes in OA tissues Recent studies have established a strong association between ferroptosis and the pathological features of OA, including cartilage degeneration, subchondral bone remodeling, and synovial inflammation

Semax altered the expression of genes that modulate the amount and mobility of immune cells and enhanced the expression of genes that encode chemokines and immunoglobulins

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