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These pathological changes were largely prevented by reducing immune cell infiltration using natalizumab, accompanied by reduced motor neuron degeneration, delayed onset of paralysis, and prolonged survival

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Strikingly, they found that in the long-lived mutants, prohibitin depletion correlates with lower fat levels, reduced mitochondria content, and increased ATP production, whereas in wild-type worms, prohibitin depletion results in only a slight decrease in fat content, an increase in mitochondrial level, and no change in ATP synthesis