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Notably, combination with anti-PD-1 checkpoint blockade yields synergy, significantly enhancing the ICB responsiveness of TNBC and producing improved therapeutic efficacy

An exhaustive investigation revealed that an augmentation in ATF4, emanating from the mTORC2-PKC-Nrf2 signaling pathway, culminates in the induction of ATF4 expression and the promotion of Sirt5, a pivotal transcriptional target of ATF4

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Liposomal and sublingual forms often provide better bioavailability